Ohwia caudata

Ethnobotanical Studies

Studies

Ohwia caudata inhibits doxorubicin-induced cardiotoxicity by regulating mitochondrial dynamics via the IGF-IIR/p-Drp1/PARP signaling pathway.

Chen JK et al (2024).
Biotechnol Appl Biochem.
PubMed:
38837810

Effective enrichment and separation of three flavonoids from Ohwia caudata (Thunberg) H. Ohashi using magnetic layered double hydroxide/ZIF-8 composites and pCEC.

Tang T et al (2024).
J Pharm Biomed Anal.
PubMed:
38714135

Ohwia caudata aqueous extract attenuates senescence in aging adipose-derived mesenchymal stem cells.

Ho TJ et al (2024).
Heliyon.
PubMed:
38698985

A combination of isoliquiritigenin with Artemisia argyi and Ohwia caudata water extracts attenuates oxidative stress, inflammation, and apoptosis by modulating Nrf2/Ho-1 signaling pathways in SD rats with doxorubicin-induced acute cardiotoxicity.

Summary

The combination of Artemisia argyi and Ohwia caudata extracts with isoliquiritigenin can protect against doxorubicin-induced cardiac damage. It activates the AKT/PI3K signaling pathway and upregulates antioxidant markers, while inhibiting inflammatory response genes. Promising for cardiac hypertrophy and inflammatory disease treatment.

Yuan Hsieh DJ et al (2023).
Environ Toxicol.
PubMed:
37661764

Ohwia caudata aqueous extract attenuates doxorubicin-induced mitochondrial dysfunction in Wharton's jelly-derived mesenchymal stem cells.

Lee PY et al (2023).
Environ Toxicol.
PubMed:
37461261

Ohwia caudata extract relieves the IL-17A-induced inflammatory response of synoviocytes through modulation of SOCS3 and JAK2/STAT3 activation.

Lu CY et al (2023).
Environ Toxicol.
PubMed:
37163279

Two New Compounds Isolated from the Leaves of Ohwia caudata and Their Neuroprotective Effect against LPS-Induced BV2 Cells.

Summary

Researchers have discovered two new compounds, along with five known compounds, in the dried leaves of Ohwia caudata. These compounds have been evaluated for their neuroprotective effect against lipopolysaccharide-induced BV2 microglia cells and have been shown to exhibit anti-neuroinflammatory activity. The structures of the new compounds were established through the use of 1D-NMR, 2D-NMR, and HR-ESI-MS spectral analysis. The previous studies conducted on O. caudata suggested that it may be capable of protecting against Alzheimer's disease.

Jin C et al (2023).
Chem Biodivers.
PubMed:
36617501

Combined effect of traditional Chinese herbal-based formulations Jing Si herbal tea and Jing Si nasal drop inhibits adhesion and transmission of SARS-CoV2 in diabetic SKH-1 mice.

Summary

In this study, researchers tested the effectiveness of two herbal-based formulations, a pleiotropic herbal drink and a nasal drop, in preventing SARS-CoV2 infection. The formulations were tested on bronchial and colorectal cell lines as well as SKH-1 mouse models, including those with comorbidities such as diabetes. The results showed that a combination of the two formulations resulted in a rapid reduction in viral entry and reduced the viral load by 97-99% in infected mice within three days of treatment. Additionally, the combination also inhibited viral transmission among cage mates. The study suggests that this combined administration could potentially prevent SARS-CoV2 infection.

Chiang CY et al (2022).
Front Pharmacol.
PubMed:
36425575

Systems Pharmacological Approach to Investigate the Mechanism of Ohwia caudata for Application to Alzheimer's Disease.

Summary

This study explored the potential of a traditional Chinese medicine, OC, for treating Alzheimer's disease. Using computer modeling, the researchers identified new compounds from OC that may improve oral bioavailability and blood-brain barrier permeability. These compounds were found to have multiple effects on AD-related pathways and processes, suggesting that they may have synergistic effects for treating AD. The study sheds light on the complex mechanisms of OC and highlights its potential as a multi-target therapy for AD.

Sun YW et al (2019).
Molecules.
PubMed:
30999553

Caudatan A, an undescribed human kidney-type glutaminase inhibitor with tetracyclic flavan from Ohwia caudata.

Sun Y et al (2018).
Phytochemistry.
PubMed:
29715600